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Univ.-Prof. Dr. med. Christoph Huber

FB 04 - Universitätsmedizin, Johannes Gutenberg-Universität Mainz

Langenbeckstr. 4, Raum: 302

  • 06131/17-72 81
  • 06131/17-66 78
Publikationen
Ergebnisse pro Seite:  10

Bobek, N.; Kuball, J.; Thaler, S. et al.

Inhibitors of apoptosis confer resistance to tumour suppression by adoptively transplanted cytotoxic T-lymphocytes in vitro and in vivo

Cell death and differentiation. D & D ; cdd. Bd. 12. H. 4. London: Arnold 2005 S. 317 - 325


Kindler, T.; Breitenbuecher, F.; Kasper, S. et al.

NEOPLASIA - Identification of a novel activating mutation (Y842C) within the activation loop of FLT3 in patients with acute myeloid leukemia (AML)

Washington, DC: American Society of Hematology 2005 S. 335 - 340


Schuler, Martin; Green, Douglas R.; Huber, Christoph

Transcription, apoptosis and p53: catch-22

Amsterdam: Elsevier 2005 S. 182 - 187


Kindler, T.; Breitenbuecher, F.; Marx, A. et al.

CLINICAL OBSERVATIONS, INTERVENTIONS, AND THERAPEUTIC TRIALS - Efficacy and safety of imatinib in adult patients with c-kit-positive acute myeloid leukemia

Washington, DC: American Society of Hematology 2004 S. 3644 - 3654



Scott, C.L.; Schuler, M.; Marsden, V.S. et al.

Research Articles - Apaf-1 and caspase-9 do not act as tumor suppressors in myc-induced lymphomagenesis or mouse embryo fibroblast transformation

New York, NY: Rockefeller Univ. Press 2004 S. 89 - 96


Schinzel, A.; Kaufmann, T.; Schuler, M. et al.

Research Articles - Conformational control of Bax localization and apoptotic activity by Pro 168

New York, NY: Rockefeller Univ. Press 2004 S. 1021 - 1032


Hasenjäger, Anne; Gillissen, Bernhard; Müller, Antje et al.

Smac induces cytochrome c release and apoptosis independently from Bax-Bcl-xL in a strictly caspase-3-dependent manner in human carcinoma cells

Basingstoke: Nature Publ. Group 2004 S. 4523 - 4535



Kindler, T.; Breitenbuecher, F.; Marx, A. et al.

CLINICAL OBSERVATIONS, INTERVENTIONS, AND THERAPEUTIC TRIALS - Brief report Sustained complete hematologic remission after administration of the tyrosine kinase inhibitor imatinib mesylate in a patient with refractory, secondary AML

Blood. the journal of hematology. Bd. 101. H. 8. Washington, DC: American Society of Hematology 2003 S. 2960 - 2962